Although the multi-collinearity among variables was evaluated, strong correlations (|correlation coefficient: r | > 0.7) were not observed. Coronary artery calcification (CAC) contributes to high risk of cardiocerebrovascular diseases in dialysis patients. Similar results have been reported with sodium phosphate enemas. However, the risk factors for CAC initiation in peritoneal dialysis (PD) patients are not known clearly. However, there are other conditions as well that might lead to the phosphate levels going out of balance. Linear regression was performed to analyze associations between the average serum phosphate level and other factors. Observational studies have determined hyperphosphatemia to be a cardiovascular risk factor in chronic kidney disease. Background: Hyperphosphatemia, serum phosphorus ≥ 4.4 mg/dL, is associated with increased risk for chronic kidney disease and cardiovascular disease. Hyperphosphatemia and hs-CRP were the independent risk factors for CAC initiation in PD patients. Renal insufficiency or renal failure is one of the common causes of hyperphosphatemia. The relative risk of overall graft loss and acute rejection among African American renal transplant recipients is attenuated with advancing age. risk factors (London et al., 2003), the search for non-traditional risk factors has led to increasing evidence of a multitude of factors that contribute to ectopic calcification in CKD. Hyperphosphatemia as an independent risk factor for coronary artery calcification progression in peritoneal dialysis patients BMC Nephrology , Jul 2015 Da Shang , Qionghong Xie , Xiaolin Ge , Huanqing Yan , Jing Tian , Dingwei Kuang , Chuan-Ming Hao , Tongying Zhu kidney-specific risk factors, such as enhanced activity of the renin–angiotensin system, sympathetic overactivity, endothelial dysfunction (related to the accumulation of asymmetric dimethylarginine, chronic inflammatory state, and oxidative stress), hyperphosphatemia, and CKD … 1.2 High mortality rate and hyperphosphatemia Inorganic phosphate (phosphate) retention, or hyperphosphatemia… In end-stage renal disease, this response becomes maladaptive and high levels of phosphorus may occur. The purpose of this study was to determine risk factors for hyperphosphatemia in ESKD patients treated with peritoneal dialysis (PD). In addition, compared with patients who had an eGFR of 60-89 mL/min/1.73 m2, those with an eGFR below 29 had a 2.3 times increased risk of hyperphosphatemia. Background. Causes of hyperphosphatemia (S) Syndrome of Tumor Lysis is a metabolic problem that mainly occurs with treatment of cancer with chemotherapy. Increased age, abnormal bowel motility, and decreased kidney function are all risk factors for persistent and significant hyperphosphatemia. Indeed, patients with hyperphosphatemia on hemodialysis have an elevated risk of lethal forms of CVD, such as myocardial infarction and stroke [7,8]. Increased age, abnormal bowel motility, and decreased kidney function are all risk factors for persistent and significant hyperphosphatemia. Methods. In advanced CKD, increased dietary phosphorus is associated with hyperphosphatemia. Hyperphosphataemia Is Associated with the Diabetes-related Cardiovascular Risk Factors Hyperphosphatemia has been associated with adverse outcomes in patients with end stage kidney disease (ESKD). The prevalence of hyperphosphatemia in the general population and in persons with renal failure is similar throughout the world.Hyperphosphatemia is a well recognized risk factor for cardiovascular mortality in dialysis patients. Hyperphosphatemia: a novel risk factor for mortality in chronic kidney disease Cardiovascular (CV) mortality in chronic kidney disease (CKD) associate with traditional risk factors (1) including dyslipidaemia, diabetes, smoking and left ventricular mass hypertrophy. Risk factors for the development of hypermagnesemia. When there is too much phosphate in the blood, the condition is known as hyperphosphatemia [1]. BMC Nephrology. Bone and mineral metabolism becomes dysregulated with progression of chronic kidney disease (CKD), and increasing levels of parathyroid hormone serve as an adaptive response to maintain normal phosphorus and calcium levels. This information will be used to develop a patient specific phosphate binder application to facilitate patient self-management of serum phosphate. Hyperuricemia and/or hyperphosphatemia were presumed responsible for the development of azotemia in the post-chemotherapy period. These results suggested potential clinical strategies to prevent the initiation of CAC in PD patients. Lower body weight and female gender: Hyperphosphatemia risk factors after sodium phosphate preparations Parakkal Deepak and Eli D Ehrenpreis Parakkal Deepak, Department of Gastroenterology, NorthShore University Hospital, 2650 Ridge Ave Evanston, IL 60201-1718, United States Hyperphosphatemia can weaken bones and cause damage to veins, tissues, and organs in the body. [Medline] . Slowing kidney damage is the best way to reduce the risk of hyperphosphatemia [2]. A peripheral form known as calcific uremic arteriolopathy (calciphylaxis) can induce necrotic ulceration and gangrene in affected extremities. This information will be used to develop a patient specific phosphate binder application to facilitate patient self-management of serum phosphate. The present study aimed to identify modifiable risk factors for... Hyperphosphatemia as an independent risk factor for coronary artery calcification progression in peritoneal dialysis patients | SpringerLink Hyperphosphatemia has been associated with increases in vessel wall thickness and arterial stiffness [24]. Conclusions. Marked elevation of phosphorus is due to these factors: Renal insufficiency – acute or chronic. Multivariate analysis revealed that hyperphosphatemia and hs-CRP were the independent risk factors for CAC initiation after adjustments. Hyperphosphatemia is commonly caused by the alteration of our renal system causing the decrease of renal excretion of phosphate. It causes the electrolytes to imbalance due to the cell dying and releasing intracellular contents into the blood, hence too much phosphate is released into the blood Clin Transplant . Hyperphosphatemia is a risk factor for adverse outcome in patients on regular hemodialysis. Previous studies have shown a weak association between dietary phosphorus … Hyperphosphatemia in adults is defined as a serum phosphorus level greater than 5.0 mg/dl. The risk factors for hyperphosphatemia. With regard to sudden death, two previous observational studies suggested an association between serum phosphate levels and the risk of sudden death in patients on hemodialysis[ 9 , 10 ]. Two patients were dialyzed for renal failure due to hyperuricemia and one for renal failure due to hyperphosphatemia which developed shortly after starting chemotherapy. 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